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Nephritis
can
be due
to
lupus,
anaphylactoid purpura, etc., and some
cases
would
not even have any asso-
ciated
systemic diseases
at
all
-
these are termed "primary"
chronic
glomerulo-nephritis. As such the dis-
ease well deserves further discussion.
Of
all
nephritis patients, those
with
swelling
are
just
a
minority
of the
whole
patient population.
lf
we merely equate
"nephritis" with "swelling", we
may
miss
a considerable
proportional
of nephri-
tis
patients.
Their presentation
is usu-
ally anaemia, too scanty or too
much
urine
output,
high blood pressure, etc.
As
the kidney
has
great reserves, once
signs
of kidney
failure
begin to develop,
the kidneys have already substantial
damage.
The occurrence
of
uncon-
trolled high blood pressure
in
the pres-
ence
of
nephritis would actually worsen
the
nephritis. A good way of detecting
kidney diseases is through
regular
health check
ups.
Recent research has
now managed
to enter
"the
era
of
cellular and molecular biology";
using
hi-tech methods,
it
would soon
be
tea-
sible to treat the untreatable and this
is
no longer scientific
fiction.
Scientific
workers
have
already employed spe-
cific antibodies against nephritis
to
block
the
kidney tissues
from
contact-
ing
immunological complexes, so
that
there would not be damage to
the
kid-
ney
tissues. This would etfectively pre-
vent
or halt
the progression of nephritis.
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