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The
occurrence of unstable
angina
pectoris
and acute myocar-
dial
infarction
is
related
to
platelet adhesion
and
aggregation,
which
cause
the
formation of thrombosis through fibrin deposition
at
the
site
of
coronary arterial
intimal injury,
and
related
to
certain
humoral mediators
such as
thromboxane,
which
is
released by
platelets. Both
thrombosis and thromboxane may interact to give
rise
to
intrinsic narrowing
in
coronary
luminal
diameter. Aspirin
could significantly suppress platelet
aggregation.
This
suggest
that
aspirin
may
play a
role
in
prevention
and
treatment
of
coronary
heart disease.
The
prevented subjects are those who have
high
risk
factors of coronary heart disease
without hypertension.
The
effec-
tive dose of
aspirin is
75-325m9
per
day.
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